Pro-inflammatory Cytokines Mediate the Epithelial-to-Mesenchymal-like Transition of Pediatric Posterior Fossa Ependymoma

Pediatric ependymoma is a brain tumor known for its pattern of recurrence. Unfortunately, there is a lack of effective chemotherapies targeting ependymoma due to a limited knowledge of how the tumor forms and grows. This shortage of treatments is due to limited knowledge about how ependymoma tumors develop and grow. . In this study, researchers reveal key drivers in the differentiation of ependymoma tumor cells. They identify NFκB, AP-1, and MYC as mediators of this cell transition.

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Pro-inflammatory Cytokines Mediate the Epithelial-to-Mesenchymal-like Transition of Pediatric Posterior Fossa Ependymoma

Previous PostRetrospective Dataset And Survey Analyses Identify Gaps In Data Collection For Craniopharyngioma And Priorities Of Patients And Families Affected By The Disease

Next PostProteogenomic Discovery of Neoantigens Facilitates Personalized Multi-antigen Targeted T Cell Immunotherapy for Brain Tumors

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